Understanding the role of inflammation in the pathogenesis and destabilization of arterial hypertension (AH), on the one hand, and on the other hand, a significant prolongation of the life of patients with rheumatoid arthritis (RA) due to the development of modern and highly effective genetically engineered biological drugs and prohypertensive effect of most antirheumatic drugs, primarily NSAIDs and glucocorticoids, further actualize the problem of RA and AH comorbidity [2, 4, 7, 8].
Analysis of the results of diurnal blood pressure profile (DBPP) makes it possible to calculate the average values of BP per day, during daytime and at night, BP variability, assess dynamics of decrease in BP at night compared to daytime, and the level of BP in the morning [1, 3, 5].
