In the scientific literature of recent years, the role of labrocytes (LC) in the pathogenesis of chronic catarrhal gingivitis (CCG) and other diseases of the oral cavity, including tumors, has been widely discussed [2, 12-14, 16, 18].

In inflammatory periodontal diseases or other pathogenic influences, all types of LC occur, the differences are only quantitative [2, 6, 14, 17]. At the same time, there are no data in the published literature on the number of LCs and their role in the development of CCG in children with dentoalveolar anomalies (DAA).